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Researchers are particularly interested in GHK-Cus gene expression modulation capabilities, which allow it to influence a vast array of biological pathways involved in tissue repair

It is a physiologic strategy

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[14] In dermal fibroblast culture and in rat wound-chamber models, GHK-Cu has been reported to modulate glycosaminoglycan and small-proteoglycan synthesis increasing chondroitin sulphate and dermatan sulphate accumulation, raising decorin mRNA and lowering biglycan mRNA i.e

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Whats happening now is like a large-scale, open-label, public human trial thats being done on these drugs, the long-term consequences of which we dont know, noted Phillips